Objective: To study the role of mechanical, infectious, and inflammatory factors inducing neuropathic attacks in hereditary brachial plexus neuropathy (HBPN), an autosomal dominant disorder ...

1 Centre for Medical Image Computing, Department of Computer Science, University College London, Gower Street, London, UK 2 Huntington’s Disease Research Centre, University College London, 2nd Floor ...

College of Pharmacy, University of Illinois at Chicago, Chicago, IL, USA Avi Ashkenazi, MD, Department of Neurology, Thomas Jefferson University, 111 South 11th ...

Department of Clinical Neurosciences, University of Cambridge Clinical School, Addenbrooke’s Hospital, Cambridge CB2 0QQ, UK N Robertson, Helen Durham Neuro-inflammatory Centre, Department of ...

Department of Clinical Neurosciences, Western General Hospital, Crewe Road, Edinburgh EH4 2XU, UK Correspondence to: Dr R Al-Shahi, Bramwell Dott Building, Department of Clinical Neurosciences, ...

7 Department of Neurology, Queen Elizabeth Hospital, Edgbaston, Birmingham, UK 8 Department of Neurology, King's College Hospital and King's College School of Medicine, London, UK Correspondence to Dr ...

3 Cleveland Clinic Lou Ruvo Center for Brain Health, Las Vegas, Nevada, USA Correspondence to Dr Jeffrey Cummings, Cleveland Clinic Lou Ruvo Center for Brain Health, 888 W. Bonneville Avenue, Las ...

Background Previous natural history studies highlighted a consistent heterogeneity of disability trajectories among individuals with primary or secondary progressive multiple sclerosis (MS). However, ...

Disease-modifying treatments for major neurocognitive disorders, including Alzheimer’s disease, Parkinson’s disease and other cognitive deficits, are among the main unmet needs in modern medicine.

Chimeric antigen receptor (CAR) T-cell therapy has revolutionised the treatment of haematological malignancies and has demonstrated efficacy in early trials for solid tumours, neurological and ...

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Background Biomarkers for predicting myelin oligodendrocyte glycoprotein antibody (Ab)-associated disease (MOGAD) clinical course are still missing. Binding capacity to a mutant MOG protein variant ...